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Hello, welcome to my blog. This blog is designed to be a virtual shop window for my work, which can be found on this site : www.creative-dreams.com and my tumblr blog : www.creative-dreams.tumblr.com. If you would like to leave a message, please contact me.
Best wishes, Karin of interest and secondary infections from the MRSA/MSSA antibiotics treatment. However, the influence of *S. aureus* infection on the host inflammatory responses were not entirely clear. In the present study, by comparing the expression levels of the inflammatory cytokines and their receptors in the absence or presence of *S. aureus* infection, we found that *S. aureus* infection attenuated the activation of inflammatory responses, but did not completely eliminate them. Therefore, our data demonstrate that *S. aureus* infection could alter the levels of inflammatory cytokines and their receptors in rats. An interesting finding is that *S. aureus* infection enhanced the expression of IL-10 and IL-6 mRNA levels at 4 hours post-inoculation, but reduced the expression of TNF-α mRNA levels and its receptors, suggesting a biphasic effect of *S. aureus* infection on the inflammatory responses in the host.
Although BPI is an important host defense factor, increased BPI levels are also associated with systemic inflammatory conditions in human subjects. In the present study, *S. aureus* infection did not alter the protein levels of BPI and BPI mRNA at 6 hours post-infection. Notably, the level of BPI mRNA was higher in the abscess tissues of rats infected with *S. aureus* than in the tissues from the animals that received control saline treatment. However, the levels of BPI protein in the control group were significantly higher than those in the *S. aureus*-infected group. This finding was in accordance with the previous report that the protein level of BPI was not up-regulated following *S. aureus* challenge, although the BPI gene was significantly induced \[[@B17]\]. These results suggested that the BPI level in *S. aureus*-infected host tissues might be regulated at the post-transcriptional level. Interestingly, the levels of BPI mRNA and protein were reduced after
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