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To study the in vivo expression and activity of peroxisome proliferator-activated receptor gamma (PPARgamma) in colorectal tumors, we generated and characterized a polyclonal antibody directed against the ligand-binding domain of PPARgamma. We also examined the expression of PPARgamma protein and mRNA in a murine model of colonic carcinogenesis. Our results show that the protein expression of PPARgamma in the tumors was 20 times higher than in the normal colonic epithelium. Interestingly, the expression and activity of PPARgamma in tumors were markedly enhanced by the administration of rexinoids to the animals. Furthermore, in vivo and in vitro characterization of the tumors revealed that they expressed high levels of mucin-type transglutaminase, a factor that may contribute to the high level of proteolytic enzyme expression in cancerous cells. Taken together, our results suggest that rexinoids may be potential drugs for the treatment of colon cancer.Q:

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